Acute gastritis
Updates to Article Attributes
Body
was changed:
Acute gastritis is a broad term for myriad causes of gastric mucosal inflammation.
Epidemiology
Depends on the aetiology: (see below).
Clinical presentation
- asymptomatic
- epigastric pain/tenderness
- nausea and vomiting
- loss of appetite
Pathology
Aetiology
- infection:
H pylorH. pylori (most common) - systemic illness: trauma and burns
- pharmacological/medication: NSAIDS
- autoimmune
- caustic ingestion
- acid
/ alkali/alkali ingestion -
immunosupressionimmunosuppression/AIDS-related:- cytomegalovirus
- candida albicans
- histoplasmosis
- cryptosporidiosis
- toxoplasmosis
- eosinophillic gastritis
Clinical presentation
asymptomaticepigastric pain / tenderness.nausea and vomitingloss of appetite
Pathology
Content pending
Radiographic features
CT
CT findings can suggest gastritis and detect complications such as gastric perforation, however, often gastritis and tumors cannot be easily differentiate on CT. Moreover, causes of gastritis cannot be determine on CT. Both circumstances require clinical/lab correlation, probable endoscopy examination and tissue biopsy1-2.
CT findings suggestive of gastritis include:
- gastric wall oedema
- measuring: measuring the HU maybe useful to differentiate oedema from low attenuation neoplastic lesion. -
ahalo signmay be present - this refers to: mucosal enhancement surrounded by submocosal and gastric wallodema.oedema
-<p><strong>Acute gastritis </strong>is a broad term for myriad causes of gastric mucosal inflammation. </p><h4>Epidemiology </h4><p>Depends on the aetiology: </p><ul>-<li>infection: H pylor</li>-<li>systemic illness: trauma and burns</li>-<li>pharmacological/medication: NSAIDS</li>-<li>autoimmune</li>-<li>caustic ingestion</li>-<li>acid/ alkali ingestion</li>-<li>immunosupression/AIDS-related : <ul>-<li>cytomegalovirus</li>-<li>candida albicans</li>-<li>histoplasmosis</li>-<li>cryptosporidiosis</li>-<li>toxoplasmosis</li>- +<p><strong>Acute gastritis </strong>is a broad term for myriad causes of gastric mucosal inflammation. </p><h4>Epidemiology </h4><p>Depends on the aetiology (see below). </p><h4>Clinical presentation</h4><ul>
- +<li>asymptomatic</li>
- +<li>epigastric pain/tenderness</li>
- +<li>nausea and vomiting</li>
- +<li>loss of appetite </li>
- +</ul><h4>Pathology </h4><h5>Aetiology</h5><ul>
- +<li>infection: <em>H. pylori</em> (most common)</li>
- +<li>systemic illness: trauma and burns</li>
- +<li>pharmacological/medication: NSAIDS</li>
- +<li>autoimmune</li>
- +<li>caustic ingestion</li>
- +<li>acid/alkali ingestion</li>
- +<li>immunosuppression/AIDS-related <ul>
- +<li>cytomegalovirus</li>
- +<li>candida albicans</li>
- +<li>histoplasmosis</li>
- +<li>cryptosporidiosis</li>
- +<li>toxoplasmosis</li>
-</li>-<li><a href="/articles/eosinophillic-gastritis" title="Eosinophillic gastritis">eosinophillic gastritis</a></li>-</ul><h4>Clinical presentation</h4><ul>-<li>asymptomatic</li>-<li>epigastric pain / tenderness.</li>-<li>nausea and vomiting</li>-<li>loss of appetite </li>-</ul><h4>Pathology </h4><p>Content pending</p><h4>Radiographic features </h4><h5><strong>CT </strong></h5><p>CT findings can suggest gastritis and detect complications such as gastric perforation, however, often gastritis and tumors cannot be easily differentiate on CT. Moreover, causes of gastritis cannot be determine on CT. Both circumstances require clinical/lab correlation, probable endoscopy examination and tissue biopsy<sup>1-2</sup><sub>. </sub></p><p>CT findings suggestive of gastritis include:</p><ul>-<li>gastric wall oedema - measuring the HU maybe useful to differentiate oedema from low attenuation neoplastic lesion. </li>-<li>a halo sign may be present - this refers to mucosal enhancement surrounded by submocosal and gastric wall odema. </li>- +</li>
- +<li><a href="/articles/eosinophilic-gastroenteritis">eosinophillic gastritis</a></li>
- +</ul><h4>Radiographic features </h4><h5><strong>CT </strong></h5><p>CT findings can suggest gastritis and detect complications such as gastric perforation, however, often gastritis and tumors cannot be easily differentiate on CT. Moreover, causes of gastritis cannot be determine on CT. Both circumstances require clinical/lab correlation, probable endoscopy examination and tissue biopsy <sup>1-2</sup><sub>. </sub></p><p>CT findings suggestive of gastritis include:</p><ul>
- +<li>gastric wall oedema: measuring the HU maybe useful to differentiate oedema from low attenuation neoplastic lesion. </li>
- +<li>halo sign: mucosal enhancement surrounded by submocosal and gastric wall oedema</li>
References changed:
- 3. Brant WE, Helms CA. Fundamentals of Diagnostic Radiology. Lippincott Williams & Wilkins. (2007) ISBN:0781761352. <a href="http://books.google.com/books?vid=ISBN0781761352">Read it at Google Books</a> - <a href="http://www.amazon.com/gp/product/0781761352">Find it at Amazon</a><span class="ref_v3"></span>
Images Changes:
Image 2 Nuclear medicine (PET-CT) ( create )
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