Crossed cerebellar diaschisis in a patient with Dyke-Davidoff-Masson syndrome
Updates to Case Attributes
MRI features of right cerebellar atrophy are most likely due to crossed cerebellar diaschisis secondary to contralateral left supratentorial cerebral infarction in childhood (Dyke-Davidoff-Masson syndrome).
Crossed cerebellar diaschisisrefers to a depression in function, metabolism, and perfusion affecting a cerebellar hemisphere occurring as a result of a contralateral focal supratentorial lesion (usually an infarct). The mechanism is thought to be an interruption of cortico-ponto-cerebellar white matter tracts, which then results in deafferentationdifferentiation and hypometabolism of the contralateral cerebellar hemisphere.
-<p>MRI features of right cerebellar atrophy most likely due to <a href="/articles/crossed-cerebellar-diaschisis" title="Crossed cerebellar diaschisis">crossed cerebellar diaschisis</a> secondary to contralateral left supratentorial cerebral infarction in childhood (<a href="/articles/dyke-davidoff-masson-syndrome" title="Dyke-Davidoff-Masson syndrome">Dyke-Davidoff-Masson syndrome</a>).</p><p>Crossed cerebellar diaschisis<strong> </strong>refers to a depression in function, metabolism, and perfusion affecting a <a href="/articles/cerebellum">cerebellar hemisphere</a> occurring as a result of a contralateral focal supratentorial lesion (usually an infarct). The mechanism is thought to be an interruption of cortico-ponto-cerebellar white matter tracts, which then results in deafferentation and hypometabolism of the contralateral cerebellar hemisphere.</p>- +<p>MRI features of right cerebellar atrophy are most likely due to <a href="/articles/crossed-cerebellar-diaschisis" title="Crossed cerebellar diaschisis">crossed cerebellar diaschisis</a> secondary to contralateral left supratentorial cerebral infarction in childhood (<a href="/articles/dyke-davidoff-masson-syndrome" title="Dyke-Davidoff-Masson syndrome">Dyke-Davidoff-Masson syndrome</a>).</p><p>Crossed cerebellar diaschisis<strong> </strong>refers to a depression in function, metabolism, and perfusion affecting a <a href="/articles/cerebellum">cerebellar hemisphere</a> occurring as a result of a contralateral focal supratentorial lesion (usually an infarct). The mechanism is thought to be an interruption of cortico-ponto-cerebellar white matter tracts, which then results in differentiation and hypometabolism of the contralateral cerebellar hemisphere.</p>