Massive encephalomalacia
Updates to Case Attributes
Follow-up examination for the patient with a history of the massive stroke.
Four years ago the patient had a massive ischemic stroke which complicated with hemorrhage and threatening subfalcine herniation and underwent urgent left craniotomy for decompression. He had a complete occlusion of the left ICA.
Afterward, he underwent the rehabilitation but the right facial nerve palsy, right side spastic hemiparesis, and motoric aphasia persist.
-<p>Follow-up examination for the patient with a history of massive stroke.</p><p>Four years ago the patient had a massive ischemic stroke which complicated with hemorrhage and threatening subfalcine herniation and underwent urgent left craniotomy for decompression. He had a complete occlusion of the left ICA.</p><p>Afterward, he underwent the rehabilitation but the right facial nerve palsy, right side spastic hemiparesis, and motoric aphasia persist.</p><p> </p>- +<p>Follow-up examination for the patient with a history of the massive stroke.</p><p>Four years ago the patient had a massive ischemic stroke which complicated with hemorrhage and threatening subfalcine herniation and underwent urgent left craniotomy for decompression. He had a complete occlusion of the left ICA.</p><p>Afterward, he underwent the rehabilitation but the right facial nerve palsy, right side spastic hemiparesis, and motoric aphasia persist.</p><p> </p>
Updates to Study Attributes
Postoperative sequelae.
There is an almost complete destruction of the left hemisphere parenchyma with massive encephalomalacia which has a signal intensity of the CSF in all sequences and mild surrounding gliosis.
There is also an area in the pons, parasagittally on the left which follows signal intensity of the CSF and also represents a smaller area of the encephalomalacia.
LeftThe left lateral ventricle is enlarged, especially the body and the posterior horn. The fourth ventricle is also enlarged and more pronounced also the third ventricle.
There is an irregularly shaped area in the right cerebellar hemisphere with hyper signal intensity on T2 and FLAIR and hypo signal intensity on T1 which probably represents an old ischemic lesion.
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