Vein of Trolard thrombosis with venous infarction (CT perfusion)

Dysphasia, dysarthria, right upper limb paresthesia and reduced power. 5 days post partum. ? CVA?CVA

No intra- or extra-axial haemorrhage. No space-occupying lesion, mass effect or midline shift evident. The ventricles and CSF spaces appear normal. 

Subtle hypodensity and loss of grey-white matter differentiation in the left preprecentral and postcentral gyri. There is a prominent hyperdense cortical vein (dense vein sign) in the region representing the superior anastomotic vein (of Trolard). No dense artery sign.

No evidence of dissection or thrombus within the carotid arteries or vertebrobasilar system. Normal appearance of the circle of Willis. No aneurysm identified. Fenestrated right A2 origin.

The previously identified cortical vein (vein of Trolard) within the left frontotemporalfrontoparietal region demonstrates no contrast enhancement in keeping with thrombosis. Normal contrast enhancement within the dural venous sinuses and internal cerebral veins.

A wedge-shaped focus of abnormal perfusion is seen within the left frontoparietal area at the vertex with decreased cerebral blood flow (CBF), increased mean transit time (MTT) and time to drain, and increased cerebral blood volume (CBV). This in keeping with a penumbra.

Cortically based gyriform restricted diffusion within the left parietal lobe (postcentral gyrus), corresponding to the perfusion abnormalities demonstrated on the CT and in keeping with acute infarction. Associated increased T2/FLAIR signal within the affected swollen gyri. An enlarged left superficial cortical vein (the vein of Trolard) shows susceptibility-weighted abnormality representing thrombosis.

Increased FLAIR signal within some parietal and frontal sulci just anterior to the infarct with associated susceptibility weighted artifact, in keeping with a small volume acute sulcal subarachnoid haemorrhage.

No further foci of abnormal restricted diffusion. Otherwise normal signal intensity of the neuroparenchyma. Neuroparenchymal volume is preserved. The ventricular system is within normal limits for age.

IMPRESSION

Acute infarct within the cortex of the left parietal lobe secondary to thrombosis of the vein of Trolard. Small volume of adjacent acute sulcal subarachnoid haemorrhage but no intraparenchymal haemorrhage.