Presentation
Progressive disturbed level of consciousness, weakness and blurred vision.
Patient Data
Bilateral nearly symmetrical hypodensity of putamina reflecting putaminal necrosis. Bilateral cerebral white matter predominantly frontal abnormal hypodensities.
Bilateral nearly symmetrical putaminal abnormal signal nearly follows CSF signal in all pulse sequences suggestive of encephalomalacia and necrosis. Cystic changes noted on the right side. Bilateral symmetrical basal ganglia abnormal high signal intensity in T2 and FLAIR. Bilateral cerebral white matter predominantly frontal cortical and subcortical patchy signal alteration elicits high signal intensity in T2 and FLAIR. Left cerebellar ill-defined patchy area of similar signal alteration.
Case Discussion
Methanol poisoning is a cause of an acute toxic leukoencephalopathy that also has eventual chronic sequelae. The patient clinical history was positive for the oral ingestion of methanol for a long time. The Putamina are the most susceptible sites for damage in methanol intoxication. Cerebral and cerebellar affection may also occur. The patient passed away after four months of hospitalization.
Unable to process the form. Check for errors and try again.